Tree Damage From Crop Spraying

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Update on trees...
The silver maple I posted a few days ago "doing nicely" not so much any more. All the new growth is withering up.
Same with the oakleaf hydrangea, poplars, paulownia, and pecan.
The photos of the leaves on the poplars is all new growth on the green stems.
The little oaks look like total crap. As soon as the inspector sees them later this week, I'll be putting them out of their misery.

maple3.JPGmaple5.JPGoakleafHydrangea1.JPGpaulownia2.JPGpecan1.JPGpoplar1.JPGpoplar2.JPGpoplar4.JPG
 
"2,4-D is an herbicide that kills plants by changing the way certain cells grow. 2,4-D comes in several chemical forms, including salts, esters, and an acid form. The toxicity of 2,4-D depends on its form. The form also affects what will happen to 2,4-D in the environment and what impacts it may have, especially on fish. 2,4-D is used in many products to control weeds, and it is often mixed with other herbicides in these products.

2,4-D kills broadleaf weeds but not most grasses. 2,4-D kills plants by causing the cells in the tissues that carry water and nutrients to divide and grow without stopping. Herbicides that act this way are called auxin-type herbicides.

One form of 2,4-D, the butoxyethyl ester, had a much longer half-life in aquatic sediment of 186 days.
Some of the ester forms of 2,4-D can be very toxic to fish and other aquatic life."
http://npic.orst.edu/factsheets/24D...2,4-D work?,are called auxin-type herbicides.

How long does the 2-4-D stay in the plant tissue and how long does it's effects last in the plant?
 
"Mode of Action: 2,4-D is an “auxin mimic” or synthetic auxin. This type of herbicide kills the target weed by mimicking the plant growth hormone auxin (indole acetic acid), and when administered at effective doses, causes uncontrolled and disorganized plant growth that leads to plant death. The exact mode of action of 2,4-D is not fully understood, and it is possible that it causes a variety of effects which are fatal when combined. It is believed to acidify the cell walls which allows the cells to elongate in an uncontrolled manner. Low concentrations of 2,4-D can
also stimulate RNA, DNA, and protein synthesis leading to uncontrolled cell division and growth, and, ultimately, vascular tissue destruction. On the other hand, high concentrations of 2,4-D can inhibit cell division and growth. Plant death typically occurs within three to five weeks following application.

Some animals such as dogs, however, are significantly more sensitive to 2,4-D organic acids than are rats and humans (Ibrahim et al. 1991). In 1991, Hayes et al. reported a significant increase in the occurrence of malignant lymphoma among dogs whose owners applied 2,4-D to their lawns."
https://www.invasive.org/gist/products/handbook/10.24-d.pdf
Well, that might explain the tumor on my dogs toe. :mad:
 
2,4-D can accumulate in fish exposed to concentrations as low as 0.05 ppm (Wang et al 1994b) and concentrations of 1.5 ppm can kill the eggs of fathead minnows in 48 hours (Thomas & Duffy 1968). After animals are removed from contaminated waters, they tend to excrete residues.
 
Application Considerations: The most volatile of the 2,4-D esters, methyl and isopropyl, have been banned in the U.S. (Que Hee & Sutherland 1981), but some volatile ester formulations of 2,4-D remain available. Both localized damage from immediate drift, and widespread damage resulting from clouds of volatilized 2,4-D, have been reported (WHO 1984). To reduce the amount lost to vaporization, low-volatile (long-chain) esters are available. In addition, the alkali and amine salts are much less volatile and may be more appropriate for use where esters could volatize and damage nontarget plants (WHO 1984). Volatilization also can be reduced by using corn oil or cottonseed oil adjuvants (WHO 1984). Spray nozzles should deliver a coarse spray and 2,4-D should not be applied when wind speeds exceed five miles per hour (Hansen et al. 1984).
 
Also problematic: 2,4-D sticks around in the environment. Depending on the formulation, it can drift through the air from the fields where it is sprayed or be tracked inside homes by pets or children. By the EPA's own measure, 2,4-D has already been detected in groundwater and surface water, as well as in drinking water. Australian scientists reported in 2012 that it was found in more than 90 percent of samples taken from agricultural catchments bordering the Great Barrier Reef—bad news for many fish, for whom the herbicide can be toxic. It can also poison small mammals, including dogs who can ingest it after eating grass treated with 2,4-D.
https://www.nrdc.org/stories/24-d-most-dangerous-pesticide-youve-never-heard
 

Mode of action​

2,4-D acts by mimicking the action of the plant growth hormone auxin, which results in uncontrolled growth and eventually death in susceptible plants.[24] It is absorbed through the leaves and is translocated to the meristems of the plant. Uncontrolled, unsustainable growth ensues, causing stem curl-over, leaf withering, and eventual plant death. 2,4-D is typically applied as an amine salt, but more potent ester versions exist, as well
 
Health effects of 2,4-D are of particular concern due to its widespread distribution and ability to drift off-site. Levels of 2,4-D have been detected in indoor air and surfaces (floors, tables, windowsills) following lawn application of the herbicide. In these instances, exposure levels for children are significantly higher than pre-application, resulting in continuous, long-term elevated exposures. Even though many believed the use of 2,4-D would gradually be reduced
and replaced by newer generations of pesticides, 2,4-D use is now expected to grow substantially in the coming years. This is because Dow AgroSciences has begun marketing corn and soybean genetically engineered to be resistant to 2,4-D, allowing farmers to spray more of the herbicide across agricultural regions.
https://www.beyondpesticides.org/assets/media/documents/pesticides/factsheets/2-4-D.pdf
 
Mode of Action 2,4-D is a selective herbicide used to kill broadleaf weeds for post-emergent control. It is a plant growth regulator, and mimics the natural plant growth hormone, auxin. 2,4-D remains at high levels within plant tissues and causes rapid cell growth. Plants die when their vascular transport systems become blocked and destroyed by abnormally fast growth. While 2,4-D is normally applied to a plant’s leaves, it can be absorbed through the roots and stems.
 
"This year, the high temperature at my farm on May 5 is in the mid 50’s and the forecast high for early next week is approaching 85 and windy. Opportunities to follow use labels for any pesticide will be scarce this year. Yet, I have grave doubts that many row crop fields will remain unsprayed.

How many times will I find renewed energy to put my food crops into the ground, knowing that chemical trespass is inevitable again this year? How long until the EPA figures out that dicamba products simply cannot be safely used because the agricultural systems and the weather make it impossible to do so?"

https://www.panna.org/blog/perfect-storm-more-dicamba-damage
 
"Herbicide Symptomology Growth-Regulator Herbicides (Hormonal Herbicides) While plants may possess differing sensitivity to various growth-regulator herbicides (hormonal herbicides), when exposed to damaging levels of these chemicals, basic symptomology is similar. Herbicides commonly encountered in this group include 2,4-D, MCPP (Mecoprop), triclopyr (Remedy), aminopyralid (Milestone), clopyralid (Transline), dicamba (Banvel), picloram (Grazon P+D), and quinclorac (Paramount). Damage severity from these herbicides depends on the concentration to which the plant has been exposed. Common lower concentration symptoms include shoot tip twisting, epinasty in foliage, chlorosis, and parallel leaf venation. Exposure to higher concentrations often results in severe chlorosis, necrosis in shoot tips and leaves, and even mortality. Leaf necrosis, dieback, and mortality are more prevalent in instances of heavy exposure to dicamba and picloram. Symptoms are prominent on new growth, and current-year twigs/shoots may be noticeably twisted and flattened."
http://extension.msstate.edu/sites/default/files/publications/publications/p3273.pdf
 
Or he/she stepped on a thorn as dogs do on a daily basis. You need to get him/her booties and yes they make them...............
You're not a veterinarian.
The vet diagnosed the tumor and removal of the tumor would involve removal of the toe and/or foot.
Now why don't you quit being an ******* and

soak your head.gif
 
Try these they seem to offer good protection and are very vogue.......
https://www.lightinthebox.com/en/p/...ping&msclkid=4880c5aee66311edef9ba73be205ac8f
And this is your response to me saying my dog has a tumor on his toe and might require his foot to be amputated??
Making light of the potential chemical poisoning of wild and domestic animals coming in contact with herbicides?

You have added nothing of value to this thread, just the opposite in fact. And that's another fact.

But know this, I've punched your "Ignore" button and from this point forward, I will no longer see any of your posts... sure-fire way of making you permanently disappear. :dancing: B-bye :hi:
 

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